When tested in mice, psoriasis became more severe when more interferon kappa was present.

The new approach blocks the interaction between cancer cells and immune receptors, showing promise in mice.

The surprising role of an immune cell that explains some of the drugs' side effects and could lead to better treatments for persistent infections and cancer.

Treatment planning for a painful and potentially disabling condition could be made easier with a new model for assessing genetic risk in people with psoriasis.

The identification of 16 additional genetic markers will help researchers get closer to understanding how — and why — psoriasis develops.